ICG  

Accounting System of Scientific Activity (ASSA)

      

Grigory Dianov

Is not an employee of the Institute
Department: Mechanisms of genome stability
Room: Дистанционная работа
Email: DianovGL@icg.sbras.ru


Publications

2021 Boron-containing nucleosides as tools for boron-neutron capture therapy
Zharkov, D. O., Yudkina, A., Riesebeck, T., Loshchenova, P. S., Mostovich, E. A. & Dianov, G. L.
[American Journal of Cancer Research]
Mild phenotype of knockouts of the major apurinic/apyrimidinic endonuclease APEX1 in a non-cancer human cell line
Kim D.V., Kulishova L.M., Torgasheva N.A., Melentyev V.S., Dianov G.L., Medvedev S.P., Zakian S.M., Zharkov D.O.
[PloS One]
2020 The role of Sp1 in the detection and elimination of cells with persistent DNA strand breaks.
Polina S. Loshchenova, Svetlana V. Sergeeva, Sally C. Fletcher and Grigory L. Dianov.
[NAR Cancer]
Sp1-independent downregulation of NHEJ in response to BER deficiency.
Loshchenova PS, Sergeeva SV, Limonov DV, Guo Z, Dianov GL.
[DNA REPAIR]
2019 E2F1 proteolysis via SCF-cyclin F underlies synthetic lethality between cyclin F loss and Chk1 inhibition.
Burdova K, Yang H, Faedda R, Hume S, Chauhan J, Ebner D, Kessler BM, Vendrell I, Drewry DH, Wells CI, Hatch SB, Dianov GL, Buffa FM, D'Angiolella V.
[EMBO J]
Two-way crosstalk between BER and c-NHEJ repair pathway is mediated by Pol-β and Ku70.
Xia W, Ci S, Li M, Wang M, Dianov GL, Ma Z, Li L, Hua K, Alagamuthu KK, Qing L, Luo L, Edick AM, Liu L, Hu Z, He L, Pan F, Guo Z.
[FASEB J]
2018 Persistent DNA strand breaks induce a CAF-like phenotype in normal fibroblasts.
Legrand AJ, Poletto M, Pankova D, Clementi E, Moore J, Castro-Giner F, Ryan AJ, O'Neill E, Markkanen E, Dianov GL.
[ONCOTARGET]
RASSF1A uncouples Wnt from Hippo signalling and promotes YAP mediated differentiation via p73.
Papaspyropoulos A, Bradley L, Thapa A, Leung CY, Toskas K, Koennig D, Pefani DE, Raso C, Grou C, Hamilton G, Vlahov N, Grawenda A, Haider S, Chauhan J, Buti L, Kanapin A, Lu X, Buffa F, Dianov G, von Kriegsheim A, Matallanas D, Samsonova A, Zernicka-Goetz M, O'Neill E,
[NAT COMMUN]
Sp1 phosphorylation by ATM downregulates BER and promotes cell elimination in response to persistent DNA damage.
Fletcher SC, Grou CP, Legrand AJ, Chen X, Soderstrom K, Poletto M, Dianov GL.
[NUCLEIC ACIDS RES]
2017 Impaired oxidative stress response characterizes HUWE1-promoted X-linked intellectual disability
Bosshard M, Aprigliano R, Gattiker C, Palibrk V, Markkanen E, Hoff Backe P, Pellegrino S, Raymond FL, Froyen G, Altmeyer M, Bjørås M, Dianov GL, van Loon B.
[SCI REP-UK]
Modulation of proteostasis counteracts oxidative stress and affects DNA base excision repair capacity in ATM-deficient cells.
Poletto M, Yang D, Fletcher SC, Vendrell I, Fischer R, Legrand AJ, Dianov GL.
[NUCLEIC ACIDS RES]
2016 DNA Damage and Repair in Schizophrenia and Autism: Implications for Cancer Comorbidity and Beyond.
Markkanen E, Meyer U, Dianov GL
[INT J MOL SCI]
p53 coordinates base excision repair to prevent genomic instability.
Poletto M, Legrand AJ, Fletcher SC, Dianov GL
[NUCLEIC ACIDS RES]
2015 Cells deficient in base-excision repair reveal cancer hallmarks originating from adjustments to genetic instability
Markkanen E, Fischer R, Ledentcova M, Kessler BM, Dianov GL.
[NUCLEIC ACIDS RES]
Inhibiting WEE1 selectively kills histone H3K36me3-deficient cancers by dNTP starvation
Pfister SX, Markkanen E, Jiang Y, Sarkar S, Woodcock M, Orlando G, Mavrommati I, Pai CC, Zalmas LP, Drobnitzky N, Dianov GL, Verrill C, Macaulay VM, Ying S, La Thangue NB, D’Angiolella V, Ryan A and Humphrey TC.
[CANCER CELL]
Репарация ДНК в опухолевых стволовых клетках как фактор развития устойчивости глиом к радиотерапии
Макушева Ю. С., Дианов Г. Л.
[Vavilov journal of genetics and breeding]
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